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PLOS ONE: MET Gene Amplification and MET Receptor Activation Are Not  Sufficient to Predict Efficacy of Combined MET and EGFR Inhibitors in EGFR  TKI-Resistant NSCLC Cells
PLOS ONE: MET Gene Amplification and MET Receptor Activation Are Not Sufficient to Predict Efficacy of Combined MET and EGFR Inhibitors in EGFR TKI-Resistant NSCLC Cells

c-Met inhibitor - Wikipedia
c-Met inhibitor - Wikipedia

Mechanisms of resistance to single-agent MET TKIs in the... | Download  Scientific Diagram
Mechanisms of resistance to single-agent MET TKIs in the... | Download Scientific Diagram

HER3 and mutant EGFR meet MET | Nature Medicine
HER3 and mutant EGFR meet MET | Nature Medicine

Sensitivity and Resistance of MET Exon 14 Mutations in Lung Cancer to Eight  MET Tyrosine Kinase Inhibitors In Vitro - Journal of Thoracic Oncology
Sensitivity and Resistance of MET Exon 14 Mutations in Lung Cancer to Eight MET Tyrosine Kinase Inhibitors In Vitro - Journal of Thoracic Oncology

MET Oncogene in Non-Small Cell Lung Cancer: Mechanism of MET Dysregula | OTT
MET Oncogene in Non-Small Cell Lung Cancer: Mechanism of MET Dysregula | OTT

MET amplification causes EGFR-TKI resistance by activating... | Download  Scientific Diagram
MET amplification causes EGFR-TKI resistance by activating... | Download Scientific Diagram

The clinical efficacy of combinatorial therapy of EGFR-TKI and crizotinib  in overcoming MET amplification-mediated resistance from prior EGFR-TKI  therapy - Lung Cancer
The clinical efficacy of combinatorial therapy of EGFR-TKI and crizotinib in overcoming MET amplification-mediated resistance from prior EGFR-TKI therapy - Lung Cancer

Multiplex RNA‐based detection of clinically relevant MET alterations in  advanced non‐small cell lung cancer - Aguado - 2021 - Molecular Oncology -  Wiley Online Library
Multiplex RNA‐based detection of clinically relevant MET alterations in advanced non‐small cell lung cancer - Aguado - 2021 - Molecular Oncology - Wiley Online Library

Mechanisms of resistance to EGFR tyrosine kinase inhibitors - ScienceDirect
Mechanisms of resistance to EGFR tyrosine kinase inhibitors - ScienceDirect

The promise of selective MET inhibitors in non-small cell lung cancer with  MET exon 14 skipping - Cancer Treatment Reviews
The promise of selective MET inhibitors in non-small cell lung cancer with MET exon 14 skipping - Cancer Treatment Reviews

Liquid biopsy uncovers distinct patterns of DNA methylation and copy number  changes in NSCLC patients with different EGFR-TKI resistant mutations |  Scientific Reports
Liquid biopsy uncovers distinct patterns of DNA methylation and copy number changes in NSCLC patients with different EGFR-TKI resistant mutations | Scientific Reports

Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant  non-small cell lung cancer? | Semantic Scholar
Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant non-small cell lung cancer? | Semantic Scholar

AXL/MET dual inhibitor, CB469, has activity in non-small cell lung cancer  with acquired resistance to EGFR TKI with AXL or MET activation - Lung  Cancer
AXL/MET dual inhibitor, CB469, has activity in non-small cell lung cancer with acquired resistance to EGFR TKI with AXL or MET activation - Lung Cancer

MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer |  Journal of Hematology & Oncology | Full Text
MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer | Journal of Hematology & Oncology | Full Text

PLOS ONE: MET Gene Amplification and MET Receptor Activation Are Not  Sufficient to Predict Efficacy of Combined MET and EGFR Inhibitors in EGFR  TKI-Resistant NSCLC Cells
PLOS ONE: MET Gene Amplification and MET Receptor Activation Are Not Sufficient to Predict Efficacy of Combined MET and EGFR Inhibitors in EGFR TKI-Resistant NSCLC Cells

Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant  non-small cell lung cancer? - Cancer Treatment Reviews
Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant non-small cell lung cancer? - Cancer Treatment Reviews

1,2,4-Oxadiazole derivatives targeting EGFR and c-Met degradation in TKI  resistant NSCLC - ScienceDirect
1,2,4-Oxadiazole derivatives targeting EGFR and c-Met degradation in TKI resistant NSCLC - ScienceDirect

Figure 2 from EGFR-TKI resistance in NSCLC patients: mechanisms and  strategies. | Semantic Scholar
Figure 2 from EGFR-TKI resistance in NSCLC patients: mechanisms and strategies. | Semantic Scholar

JCM | Free Full-Text | The Role of MET Inhibitor Therapies in the Treatment  of Advanced Non-Small Cell Lung Cancer | HTML
JCM | Free Full-Text | The Role of MET Inhibitor Therapies in the Treatment of Advanced Non-Small Cell Lung Cancer | HTML

Reciprocal relationship between MET amplification and T790M mutation.... |  Download Scientific Diagram
Reciprocal relationship between MET amplification and T790M mutation.... | Download Scientific Diagram

Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant  non-small cell lung cancer? - ScienceDirect
Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant non-small cell lung cancer? - ScienceDirect

MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer |  Journal of Hematology & Oncology | Full Text
MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer | Journal of Hematology & Oncology | Full Text

Alterations in the PI3K Pathway Drive Resistance to MET Inhibitors in NSCLC  Harboring MET Exon 14 Skipping Mutations - Journal of Thoracic Oncology
Alterations in the PI3K Pathway Drive Resistance to MET Inhibitors in NSCLC Harboring MET Exon 14 Skipping Mutations - Journal of Thoracic Oncology

PDF] EGFR and c-Met Inhibitors are Effective in Reducing Tumorigenicity in  Cancer | Semantic Scholar
PDF] EGFR and c-Met Inhibitors are Effective in Reducing Tumorigenicity in Cancer | Semantic Scholar

Osimertinib plus savolitinib in patients with EGFR mutation-positive, MET-amplified,  non-small-cell lung cancer after progression on EGFR tyrosine kinase  inhibitors: interim results from a multicentre, open-label, phase 1b study  - The Lancet Oncology
Osimertinib plus savolitinib in patients with EGFR mutation-positive, MET-amplified, non-small-cell lung cancer after progression on EGFR tyrosine kinase inhibitors: interim results from a multicentre, open-label, phase 1b study - The Lancet Oncology